Friday, May 16, 2014

Burzynski Research

Burzynski Research

Long-term survival of high-risk pediatric patients with primitive neuroectodermal tumors treated with antineoplastons A10 and AS2-1.


Abstract

Primitive neuroectodermal tumors (PNETs) are usually successfully treated with craniospinal radiation and chemotherapy; however, difficulties with standard treatment can be encountered in very young children, in adult patients at high risk of complication from standard treatment, and in patients with recurrent tumors. Thirteen children, either with recurrent disease or high risk, were treated in phase II studies with antineoplastons (ANP). The median age of patients was 5 years, 7 months (range, 1-11). Medulloblastoma was diagnosed in 8 patients, pineoblastoma in 3 patients, and other PNET in 2 patients. 

Previous treatments included surgery in 12 patients (1 had biopsy only, suboccipital craniotomy), chemotherapy in 6 patients, and radiation therapy in 6 patients. Six patients had not received prior chemotherapy or radiation. The treatment consisted of intravenous infusions of 2 formulations of ANP, A10 and AS2-1, and was administered for an average of 20 months. The average dosage of A10 was 10.3 g/kg/d and of AS2-1 was 0.38 g/kg/d. Complete response was accomplished in 23%, partial response in 8%, stable disease in 31%, and progressive disease in 38% of cases. Six patients (46%) survived more than 5 years from initiation of ANP; 5 were not treated earlier with radiation therapy or chemotherapy. The serious side effects included single occurrences of fever, granulocytopenia, and anemia. The study is ongoing and accruing additional patients. The percentage of patients' response is lower than for standard treatment of favorable PNET, but long-term survival in poor-risk cases and reduced toxicity makes ANP promising for very young children, patients at high risk of complication of standard therapy, and patients with recurrent tumors.

Stanislaw R. Burzynski, MD, PhD: novel cancer research and the fight to prove its worth. Burzynski SR.
Altern Ther Health Med. 2012 May-Jun;18(3):54-61. No abstract available.
PMID: 22875562 [PubMed - indexed for MEDLINE]
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Targeted therapy with antineoplastons A10 and AS2-1 of high-grade, recurrent, and progressive brainstem glioma.
Burzynski SR, Janicki TJ, Weaver RA, Burzynski B.
Integr Cancer Ther. 2006 Mar;5(1):40-7.
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Long-term survival of high-risk pediatric patients with primitive neuroectodermal tumors treated with antineoplastons A10 and AS2-1.
Burzynski SR, Weaver RA, Janicki T, Szymkowski B, Jurida G, Khan M, Dolgopolov V.
Integr Cancer Ther. 2005 Jun;4(2):168-77.
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Phase II study of antineoplaston A10 and AS2-1 in children with recurrent and progressive multicentric glioma : a preliminary report.
Burzynski SR, Weaver RA, Lewy RI, Janicki TJ, Jurida GF, Szymkowski BG, Khan MI, Bestak M.
Drugs R D. 2004;5(6):315-26.
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Long-term survival and complete response of a patient with recurrent diffuse intrinsic brain stem glioblastoma multiforme.
Burzynski SR, Lewy RI, Weaver R, Janicki T, Jurida G, Khan M, Larisma CB, Paszkowiak J, Szymkowski B.
Integr Cancer Ther. 2004 Sep;3(3):257-61.
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Managing social conflict in complementary and alternative medicine research: the case of antineoplastons.
Hammer MR, Jonas WB.
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The present state of antineoplaston research (1).
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Integr Cancer Ther. 2004 Mar;3(1):47-58. Review.
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Phase II study of antineoplaston A10 and AS2-1 in patients with recurrent diffuse intrinsic brain stem glioma: a preliminary report.
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Potential utility of antineoplaston A-10 levels in breast cancer.
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Efficacy of antineoplastons A10 and AS2-1.
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Mayo Clin Proc. 1999 Jun;74(6):641-2. No abstract available.
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Inhibitory effect of antineoplaston A10 and AS2-1 on human hepatocellular carcinoma.
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Potential of antineoplastons in diseases of old age.
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Cellular accumulation of antineoplaston AS21 in human hepatoma cells.
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Toxicological study on antineoplastons A-10 and AS2-1 in cancer patients.
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The influence of antineoplaston A5 on particular subtypes of central dopaminergic receptors.
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The influence of antineoplaston A5 on the central dopaminergic structures.
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Treatment of hormonally refractory cancer of the prostate with antineoplaston AS2-1.
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Drugs Exp Clin Res. 1990;16(7):361-9.
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Stereochemical modelling studies of the interaction of antineoplaston A10 with DNA.
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Chemo-surveillance: a novel concept of the natural defence mechanism against cancer.
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Quantitative assay of plasma and urinary peptides as an aid for the evaluation of cancer patients undergoing antineoplaston therapy.
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N,N'-disubstituted L-isoglutamines as novel cancer chemotherapeutic agents.
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Drugs Exp Clin Res. 1987;13 Suppl 1:57-60.
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Chemoprevention by antineoplaston A10 of benzo(a)pyrene-induced pulmonary neoplasia.
Kampalath BN, Liau MC, Burzynski B, Burzynski SR.
Drugs Exp Clin Res. 1987;13 Suppl 1:51-5.
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Pharmacokinetic study of radioactive antineoplaston A10 following oral administration in rats.
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Drugs Exp Clin Res. 1987;13 Suppl 1:45-50.
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Phase I clinical studies of antineoplaston A5 injections.
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Tissue culture and animal toxicity studies of antineoplaston A5.
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Phase I clinical studies of antineoplaston A3 injections.
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In vitro cancer growth inhibition and animal toxicity studies of antineoplaston A3.
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Initial clinical study with antineoplaston A2 injections in cancer patients with five years' follow-up.
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Altered methylation complex isozymes as selective targets for cancer chemotherapy.
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Chronic animal toxicity studies on antineoplaston A2.
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3-Phenylacetylamino-2,6-piperidinedione, a naturally-occurring peptide analogue with apparent antineoplastic activity, may bind to DNA.
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Toxicology studies on antineoplaston A10 injections in cancer patients.
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Preclinical studies on antineoplaston A10 injections.
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Toxicology studies on antineoplaston AS2-1 injections in cancer patients.
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Toxicology studies on antineoplaston AS2-5 injections in cancer patients.
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Preclinical studies on antineoplaston AS2-1 and antineoplaston AS2-5.
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Antineoplastons: history of the research (I).
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Antineoplaston A in cancer therapy. (I).
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